Graduate Program

Biosystems


The program arose from a necessity to train professionals who can project, model and characterize various biological systems to support knowledge generation and sustainable industrial production.

Program goals: 1) training of qualified human resources to conduct teaching, research, and scientific and technological development; 2) enable the development of research and extension projects designed to enhance and expedite scientific knowledge in the field of Biological Sciences; 3) meet the demand for professionals at regional, national, and international levels.

The professional prepared and trained by the Biosystems program must hold an interdisciplinary background, being able to interlace several concepts related to the study of biological systems, not only in homeostatic conditions (cellular or systemic), but also during the development of pathogenic processes, including the study of new approaches related to the treatment of such conditions.


Research Areas:

Structure and Function;

Modeling and Technology;


Levels:

Master’s and Doctor’s degrees


Selection process:


Call for application usually opens in February and September.

More information about admission requirements and the application process

are available on the program website.


Classes Begin:

Usually in February and May (depending on the year’s calendar).

Fees:

none


Scholarship:

Limited availability, depending on case-by- case analysis.


Contact:

Website: biossistemas.ufabc.edu.br

E-mail: ppg.biossistemas@ufabc.edu.br


Program Website

Additional information about calls, application and deadlines, please go to the program Website

Faculty

NameResearch Areas
Amedea Barozzi Seabra
Ana Carolina Santos De Souza Galvão
Antonio Sergio Kimus Braz
Cesar Augusto Joao Ribeiro
Daniel Carneiro Carrettiero
Daniele Ribeiro De Araujo
Giselle Cerchiaro
Luciana Campos Paulino
Luciano Puzer
Luis Paulo Barbour Scott
Luiz Roberto Nunes
Marcela Sorelli Carneiro Ramos Impact of inflammatory response induced by ischemic renal injury on cardiac trophism; Contribution of the complement system in the modulation of cardiac hypertrophy induced by ischemic renal damage; Role of oxidative stress in cardiac hypertrophy induced by ischemic renal injury; Contribution of apoptosis in the modulation of cardiac trophism induced by ischemic renal injury
Marcelo Augusto Christoffolete
Marcia Aparecida Sperança
Maria Cristina Carlan Da Silva
Rodrigo Luiz Oliveira Rodrigues Cunha
Sergio Daishi Sasaki
Tiago Rodrigues
Wanius Jose Garcia Da Silva